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New School Of thought 2019-2020

All LDL is not the same. LDL-A is a buoyant, fluffy molecule that does no harm whatsoever as long as it is not damaged by oxidation (a process caused by free radicals that enables cholesterol to form plaque). LDL-B is a small, hard, dense, molecule that promotes atherosclerosis.

A pattern of high LDL-A is the most beneficial. Blood tests today are able to measure the number of LDL-A and LDL-B particles in a blood sample.

It is not the LDL that causes arterial plaque formation; it is the total number of particles and the size of the particles within the LDL molecule. The LDL-B particles are referred to as the small dense LDL, sdLDL.

As the total number of particles (LDL-P) increases, especially the type B (sdLDL), plaque formation increases. 

 

About 610,000 people die of heart disease in the U.S. every year — 1 in every 4 deaths.[1]

Heart disease is the leading cause of death for both men and women. More than half of the deaths due to heart disease in 2009 were in men.[1]

(CHD) is the most common type of heart disease, killing over 370,000 people annually.[1]

Every year, about 735,000 Americans have a heart attack. Of these, 525,000 are a first heart attack and 210,000 happen in people who have already had a heart attack.[2]

In a 2005 survey, most respondents — 92% — recognized chest pain as a symptom of a heart attack. Only 27% were aware of all major symptoms and knew to call 9-1-1 when someone was having a heart attack. [3] About 47% of sudden cardiac deaths occur outside a hospital. This suggests that many people with heart disease don’t act on early warning signs.[4]eart attacks have several major warning signs and symptoms:

  • Chest pain or discomfort

  • Upper body pain or discomfort in the arms, back, neck, jaw, or upper stomach

  • Shortness of breath

  • Nausea, lightheadedness or cold sweats [5]

Figure 1 shows the leading causes of death in the U.S. and for women.

Exercise, optimal nutrition with fruits and vegetables, and moderate alcoholic and smoking cessation reduce the risk of myocardial infarction by 80%. [6]

Research from the Atherosclerosis Risk in Communities (ARIC) Study has found that almost half (45%) of heart attacks appear to be clinically silent — that is, not associated with any symptoms at all, at least that the patient can recall. These sorts of silent heart attacks were picked up by use of a routine electrocardiogram (ECG). And these silent heart attacks weren’t just a meaningless abnormality picked up on a test. The silent heart attacks were associated with a similar risk of subsequent death as clinically detected heart attacks. [7.]

“The traditional evaluation, prevention and treatment strategies for the TOP FIVE CHD risk factors still result in a CHD gap. This means that about 50% of patients still have CHD or MI despite ‘normal’ levels of these risk factors as presently defined in the medical literature. There are important details within each of these TOP FIVE risk factors that are not being measured by physicians and are thus ignored in the prevention and treatment of CHD.”[8,9,10]

Only 10-15% of patients with coronary heart disease lack any of the five conventional risk factors of hypertension, hyperlipidemia, diabetes, smoking and obesity.[11]

Most healthcare professionals still order the basic cholesterol panel as shown in Figure 2. That panel has been available since black and white television, but is it adequate to determine if someone is at risk for cardiac disease?

Cholesterol: What You Need To Know

Cholesterol is a minor player in heart disease.
Cholesterol levels are a poor predictor of heart disease.
Half the people with heart disease have normal cholesterol.
Half the people with elevated cholesterol have healthy hearts.
Lowering cholesterol has extremely limited benefits. *

Current research has determined that the risk of an individual developing heart disease is more accurately determined by advanced lipid testing such as shown in Figure 4.
But this extensive testing takes longer to explain to patients, and it requires a more extensive knowledge of lipid metabolism and greater knowledge of biochemistry.

The theory that fat and cholesterol cause heart disease became widely accepted despite much evidence to the contrary. This evidence needs to be reexamined. The case needs to be reopened.

Many doctors did not agree with the cholesterol myth and questioned the science on which it was based. The studies upon which the cholesterol myth was based were later found to be problematic. The adoption of the cholesterol myth by mainstream organizations and the government had a strong political component to it. *

Cholesterol is the parent molecule for sex hormones (estrogen, progesterone and testosterone) as well as Vitamin D and bile acids needed for digestion. The only time cholesterol is a problem is if it’s oxidized (damaged), as it then sticks to the lining of the arteries and begins the process of inflammation.